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mutations and complicated kar yotype. It follows a linear evolution from the CLL clone throughout the recurrent acquisition of CDKN2A

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れたかを表しており，円が小さいほどその地点で判別され た回数は少なくなる．グラフから，設置したビーコンの付

mutations, in whom rituximab seems to obtain very little added value.59 Other genomic subgroups, which include patients with BIRC3

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Modern molecular scientific tests have delivered lots of insights into your procedures that govern the event and development of CLL, like a lot of novel mutated genes clustered in several practical pathways. The CLL epigenome is reprogrammed throughout the modulation of regulatory regions that show up de novo

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